Ganirelix Acetate peptide API

Et Pulsatile release of Gonadotropin-releasing Hormone (GNRH) Stimulat in synthesis et secretio LH et FSH. Frequency de lh pulsu in medium et nuper follicular augmenta est circa I per horam. Haec pulsus reflectitur in transiens resurget in Serum LH. Per medium menstruatae, in mole release of GNRH causat a surge LH. Midmenstrual lh surge potest trigger aliquot physiologica respondeo, comprehendo: ovulation, oocesttic meiotic resumpation, et corpus luteum formation. Formatio Corpus luteum causat Serum PROGVester levels ad oriri, dum estradiol campester cadunt. Ganirelix acetate est a GNRH Anagonist quod competitively cuneos GNRH receptores in pituitam gonadotrophs et subsequent traduction meatus. Facit celeri, reversible inhibitionis Gonadotropin secretionem. Inhibitory effectus Ganirelix acetate in pituitary LH secretio fuit fortior quam in FSH. Ganirelix acetate defecit inducere primo release of endogenous gonadotropins, consistent cum antagonism. Complete convaluisset de pituitam LH et FSH campester occurrit in XLVIII horas post Ganirelix acetate esset discontinuata.